BPC-157 is derived from a protective protein found in the stomach but is considered "synthetic" since this peptide does not exist by itself in nature. It’s touted for a wide variety of reasons and talked about for both nootropic (brain boosting) and ergogenic (performance enhancing) purposes. And based on all the studies published so far, BPC-157 is a surprisingly good regenerative agent.
Fibroblasts are a specialized type of cell found in connective tissues. These cells are involved in the creation of collagen.
The use of BPC-157 is thought to speed up the growth and spread of fibroblasts and to increase rates of oxidative resistance. In addition to its effects upon fibroblast production. It also affects F-actin formation (which is involved in the spreading of fibroblasts).
The peptide has also been successfully used to increase the rate of collagen reformation following surgery.
The above findings go a long way to explaining how the administration of BPC-157 is able to promote healthy tendon and ligament healing.
As gastrectomy is related to derogatory bone conditions such as osteoporosis it is no surprise that the use of BPC-157, with its well-known fracture and wound healing aspects, is associated with improved bone health.
Research using rabbits has shown that the peptide is able to significantly improve healing related to osteoperiosteal bone defects over a six-week (1.5 months) period, especially following an autologous cortical graft or the local application of bone marrow.
Furthermore, the number of animals that healed was much greater in the experimental than what occurred in the control group. So it seems that the use of BPC-157 can both increase the rate and the frequency of recovery from bone damage.
Although the above experiments took place in lagomorphs the authors are very excited about its potential use in the management of bone-related impairments that may occur in human patients.
One of the most exciting aspects of BPC-157 is its potential for protecting the intestines from inflammatory damage.
When toxins were given to rats to mimic the effect of intestinal inflammation, the use of this peptide reduced the number of visual markers known to be associated with intestinal gut damage.
The direct injections of BPC-157 into specific parts of rats is thought to help repair intestines through impacting nitric oxide signalling.
It can also help to overcome problems related to short gut (Short Bowel Syndrome: SBS). This condition often leads to malnutrition and dehydration because of increased incidence of diarrhoea. The use of this peptide can help to make this problematic condition much better. It is also thought to be able to help repair damaged tissues caused by inflammatory bowel disease (IBD).
This peptide is known to impact dopamine levels, though there is as yet no evidence for the direct binding of dopamine receptors. It is therefore thought that its main action with regards to dopaminergic neurotransmission is likely to be through the antagonism of dopamine itself, and this interaction is thought to reduce the effects of amphetamine on compulsive behaviours.
Although they act great as painkillers one of the big problems with NSAIDs is that they can be toxic to the gut, this frequently leads to ulcers and an irritation of the bowels.
BPC-157 is able to act as an anti-ulcer peptidergic agent and has been put forward as an NSAIDs antidote as it is beneficial against damage caused by mediated lesions in the gastrointestinal tract, brain, and the liver, and is able to counteract symptoms associated with the taking of aspirins, such as bleeding.
The present research suggests that BPC-157 does not have toxic effects itself and is thus thought likely to have very high safety in its use against gastric damage caused by NSAIDs.
As BPC-157 is able to reduce cell damage in the hippocampus of rats it very likely confers neuroprotective effects. It can help protect the brain tissue from damage when rats are given cuprizone (a toxin that scientists use to mimic the effects of schizophrenia and multiple sclerosis) it is thought that actual protection of the nerves may actually occur through intestinal processes.
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